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Study Suggests Improved Treatments for Neuropathic Pain
Thursday, Jun 26, 2008
Two chemicals associated with neurodegeneration and inflammation play important and distinct roles in development of neuropathic pain, a new study shows.  The findings may lead to new treatments that can stop neuropathic pain from developing and alleviate it after it begins.

Huntington’s Disease Protein Affects Nerve Signaling; Study Suggests New Treatments
Thursday, Jun 26, 2008
The abnormal protein found in Huntington’s disease (HD) leads to an unusually large amount of nerve signaling early in the disease process, before other problems appear, a new study shows. Partially blocking these nerve signals prevents neuron death and loss of motor function in fruit flies models of HD. The findings suggest possible new ways of delaying the onset or slowing the progression of the disease.

Nanotech Treatment Shows Promise against Spinal Cord Injury in Mice
Monday, May 19, 2008
In experiments on mice, scientists have shown that an injectable nanotech-based polymer stimulates axons to regrow all the way across a spinal injury.

Combining MRI and PET Could Yield Dynamic Pictures of the Brain
Friday, Apr 25, 2008
In experiments on mice, scientists report that they have successfully combined two brain imaging techniques – magnetic resonance imaging (MRI) and positron emission tomography (PET).

New Mouse Model for Neurofibromatosis Yields Insights into Disease Process and Treatment
Monday, Apr 21, 2008
In a move expected to enhance the development of therapies for neurofibromatosis type 1, scientists have created an improved mouse model for the disease.

Early Treatment Prevents Full-Blown Epilepsy in Animals
Friday, Mar 14, 2008
For the first time, researchers have shown that treating epilepsy-prone animals with an anticonvulsant drug prior to the development of chronic epilepsy can significantly reduce the number of seizures the animals experience, even after the treatment stops.  The study provides hope that researchers may eventually be able to prevent epilepsy in people who are at risk of the disorder because of genetic mutations or other factors.



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